Thesis2, edited

 KeithCampbell,@KeithCa41469726

Blog at www.strivetospin.com

🏴󠁧󠁢󠁳󠁣󠁴󠁿Thesis 2🏴󠁧󠁢󠁳󠁣󠁴󠁿

February 15th 1981 to ??/2019

Thesis part 1:"The reason/the repair with TKMS:TheKineticMomentialSystem, leaves many questions, unrealised yet. 

The healed Occipital burst, from childhood, has left, FP:Friction Points, in contact with the brain.

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Thalamus, and Hypothalamus,flow has been altered by the initial drop of BM, meaning flow changes,then flow restriction, at dx time. 

The friction, which is viewed on my Mri image, and I believe it to be the same for all, was caused by the calcified area, of that damaged OB:Occipital Bone, at childhood. 

 In addition, the change to blood flow, of Thalamus and HT: Hypothalamus,means  that flow would worsen due to it "furring up", over time. 

These two are what you have issues with, leading to DX time, decades later.

 We had an increasingly injured brain, as a child , waiting on the 'furring up' to happen. My TBI  day was 15/2/81.

I was 6yr 11mth old. 😯😫🤐🤦‍♂️

I forgot it, as you have. 

Till I then injected 17.5 MTX:Methotrexate. 

Then, a kid(8),memorised day, appeared, over 3 injection.

I say this after being dx @ 29. This is bang on the average age. 

. 

A REPAIR is possible. 

The MOI means the body eventually has sensory confusion(Thalamus blog) and can't deal with heat, and others, too. . 

Thalamus and Hypothalamus:HT had flow altered, regardless of the MOI, by the BM drop of Pons anyways, when Occipital leak/burst/healed gap(?) happened.The need to improve flow, of blood, to Thalamus, while lifting BM:Brain Mass off of the Diencephalon, can be achieved by using  CF:Centrifugal Force. 

HT is underneath it, but smaller.  Filling HT is going to happen prior, to Thalamus being filled, and lifting BM. (neuro god opinion need) 

The Dangers

The danger here is aperture length, of TKMS.  A causative injury may result if an MSer were subjected to CF at the wrong aperture distance, revs and angle,perhaps. 

However, if Thalamus were to be functioning, then cerebellum can't reach the friction point!

What I'm not sure of, is, will Thalamus stay inflated, and will the furred up loosened debris cause vein blockage or heart issues?

(I know we've all been unwell for years. This means our Thalamus and Hypothalamus have flattened. Therefore, with the pressure rise to unfurl them, removed, they may want to return to the "memory" shape)

However 

There's been mention of Asprin being a possibility to treat MSers. They don't know why from thing I've seen. 

I know.🤔😏😆

Asprin thins the blood. This equals better flow of a flattened Thalamus. 😁

Better flow, means less chance of a Thalamus , secondary collapse, when it's filled with, blood. 

It would therefore be a good idea to take Asprin an hour prior to TKMS.

The Methodology of Repair, using TKMS:THEKINETICMOMENTIALSYSTEM. 

The COR:Centre Of Rotation has zero Centrifugal Force, potential. 

Ie if an MSer were stood on it spinning, they'd get dizzy. No CF would occur. 

If they stood less than  1 metre away from COR, and then rotated, they'd be thrown to the outer edge, ala "Sticky Wall" fairground ride. 

If they were positioned and secured, on a Racer like frame midforce , near to COR, making a 90° bend with the head outermost, their blood would fill both hemisphere, Thalamus and Hypothalamus. This is in effect pumping the BM, or the top slice of bread in the sandwich analogy, off of it. [blogged previously) 

This filling has to be done cumulatively. This would avoid a causative injury. 

If they were to stand, a full 1 metre away from COR, and make the same 90°, more CF would occur. 

If it were 2mtrs distance, even more so again!

Its finding the safe pressure of veins,  and Diencephalon awareness(check) 

*(need vascular dr opinion+heart Dr+Neurogod) 

Think of Thalamus as a mini shape  brain. It's a chamber filled with blood. It has 4 blood inputs stemming flow to effect their output functions. 

Once impacted, all Thalamus results are different, have individual variance, greatly impact the TBI:Traumatic Brain Injury sufferer life and its all downwards, in the main, for the rest of their days. (done 5th May). 

This is literally true. 

Progression

Progression, is down to gravity. The cerebellum makes its way over the calcified  burst, made  so as a kid, then each head motion takes a tiny part of their BM.

 

Over time, more brain is impacted.

Thalamus is furring up after your OB burst, as is Hypothalamus, until dx, decade's later. 

Reasoning

I know the difference of the big 2,is down to the  magnitude of the bursting force, that separated Occipital Bone, as a kid. 

 

This means for PP, a larger force causing instantaneous opening of the midseam of OB,then a harder compression of Thalamus, which is then meaning more healed FPs, due to that wider OB burst. 

This, would need a larger aperture distance, to repair it, by filling Hypothalamus first, then a cumulative filling of Thalamus. 

The brain, for all, was RR to start with. Depending on the MOI, means how hard BM is then rubbed off, by the skull, plus the FP burst of course. 

Its the time it takes for your cerebellum to possibly reach SC:Spinal chord, that determines your journey, eventually. 

MSer day

2 primary, 2 secondary and 2 RR….on Racer like frames, mounted, and secured, on TKMS. 

Hexagon shape around the COR, with they 6 MSers, would be, OMFG! 

They completely secured MSers, WOULD KEEP THEIR FOREHEAD ON HANDLEBARS, MAKING THEIR SPINE PARALLEL TO THE DECK OF TKMS . Adjustments if needed, as this is Important.  

Varying aperture. Varied revs, would equal learning the ideal distance, and they all important  revs.

I believe each TBI will be the same. Its the damage done to Occipital Bone, and the resultant  impact force to Thalamus,, due to BM drop, that decides how to repair it, by  aperture distance. 

. 

. Ie: That impact to OB, will be proportional to the Thalamus, flattening, force. 

Perhaps, a Primary 2mtrs and Secondary 1mtr, to give you my pre assumed gameplan. This equates to my thinking a Primary Patient had a bigger Occipital burst than eventual Secondary. This bigger initial impact has likely, really flattened Thalamus and would need a bigger, initially at least, force, hence the bigger distance from COR, to start with. 

Woopdiddlydo, 

Ma grief is now true, 

"Gonnae gies a shot". 

"Is ma name in the pot?" 

When will Keith learn, 

The results we yearn? 

 

The forces, to enable filling of both Thalamus and Hypothalamus(same forces lifting BM at both sides), have to be equal. 

I also think, the direction of rotation, changing midway of the time frame, would have to happen,  too. 

Eventually momentum via CF, would make you form this tall shape. It is at they revs, we are close to the inertia needed to START to lift BM off Diencephalon and filling Thalamus, with blood. 

This is where times comes in. If you were kept at higher revs "Rx" for too long, you'd cause a causative injury . 

For sure. 🙏

The speed and aperture distance? I need to find CF percentage against distance from COR. 

This is revision. I know such a diagram exists, as I seen it at Nautical College! 👍

Must avoid causative. Must not pong. 

Research needed. (done 12th May) 

Perhaps, the top slice of BM has to be lifted by inertia, to enable Thalamus/Hypothalamus to inflate? 

TESTING

The ability to deal with heat, would be first to show an improvement via Hypothalamus. 

Hypothalamus, is a guide here.ie its max pressure, or revs, is the overall max, when Thalamus is filled, with blood. 

Hot bath monitoring at R revs, then R1,  then R2 and so on. 

Cover that wae, when the MSer gets worse, in the hot bath, or the temperature even. Then after several, you'd get a change, THAT says Hypothalamus is WORKING. 

We would then have "Rx", which becomes the cumulative filling revs for Thalamus. 

Testing, is going to be excitingly birly, wet  drama. 

I'd bet my life, on this. 

I probably will!😆👍🤔☎️😳🤗👏👏🐸….  

Conclusion

. 

My thinking is something positive would be damn quick,but if its ridiculously easy, then a causative injury is close, too. (19 may) 

I have lived this. All MSers have, in some guise.

I intentionally memorised that time, at 8 years old… 

I forgot it as you have, but I then injected 17.5mg  MTX, for Psoriatic Arthritis.

 This has enabled the minding of the memorised day fae hell. ||||||💔😣🙏🏍|||||🛫⚓🔧🏴󠁧󠁢󠁳󠁣󠁴󠁿🗺️|👩‍❤️‍💋‍👩👰🤰❤️🐎👨‍🔧

 I've then proven my kid injury with functions of Thalamus and Hypothalamus, on my blog, at www.strivetospin.com. 

.

I think this, Thalamus data, was missed. MSers numbers, are growing. Reviewing, what they truly know, hasn't  happened, due to their work load probably. 

 At present we have a reactionary system that is, Pharma swayed. Lives are then misguided, hopes are spoiled,their life paint, is tainted. 

NEEDLESSLY

The MS World needs a great big rocket, in the proverbial arse. 

I'm the lighter, of that rocket. 🦖♨️🚀

We need to OWN , MS!👨‍🔧

"It is an injury primarily, that manifests as a disease, eventually". 

"We are treating they diseases, and have missed the Pink Occipital Elephant,of a childhood cause". 

I was blown away, when this door reopened. Tears, scared, missed, pissed,  thinking, Owing, rethinking ……………………….. ……..Owning.

EVENTUALLY, I felt enormously proud, of, wee Keith. I'm fulfilling his hopes, even. 

I've learned, lots, both as an engineer, and about us, getting to here. 

I have now shared the truth, but in the main I've shown the methodology ,of  how to repair this TBI, named MS.

. 

. 

 It should be COBer for

"Collapsed Occipital Boner", btw😜. 

"Fate", is ma mate. Let me introduce yies to him.

           Honest. Learned.Lived. Loved

                        + Shared. 

Ma Mate Is nearing, 

I can hear him cheering.

" TKMS TKMS TKMS TKMS" 

Thanks for your time, 

Keith Campbell, on the 23rd May, 2019.